Histone methyltransferase G9a diminishes expression of cannabinoid CB1 receptors in primary sensory neurons in neuropathic pain-Reference-Cited by-同舟云学术

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Histone methyltransferase G9a diminishes expression of cannabinoid CB1 receptors in primary sensory neurons in neuropathic pain

Published:2020-03 Issue:11 Volume:295 Page:3553-3562
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Luo Yi,Zhang Jixiang,Chen Lin,Chen Shao-Rui,Chen Hong,Zhang Guangfen,Pan Hui-Lin

Funder

HHS | NIH | National Institute on Drug Abuse

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference63 articles.

1. Structure of a cannabinoid receptor and functional expression of the cloned cDNA;Matsuda;Nature,1990

2. Molecular characterization of a peripheral receptor for cannabinoids;Munro;Nature,1993

3. The synthetic cannabinoid WIN55,212-2 attenuates hyperalgesia and allodynia in a rat model of neuropathic pain;Bridges;Br. J. Pharmacol,2001

4. The role of central and peripheral Cannabinoid1 receptors in the antihyperalgesic activity of cannabinoids in a model of neuropathic pain;Fox;Pain,2001

5. The analgesic effects of R(+)-WIN 55,212-2 mesylate, a high affinity cannabinoid agonist, in a rat model of neuropathic pain;Herzberg;Neurosci. Lett,1997

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3. Euchromatin histone-lysine N-methyltransferase 2 regulates the expression of potassium-sodium-activated channel subfamily T member 1 in primary sensory neurons and contributes to remifentanil-induced pain sensitivity;Brain Research Bulletin;2024-06

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