Receptor for advanced glycation end products mediates sepsis-triggered amyloid-β accumulation, Tau phosphorylation, and cognitive impairment-Reference-Cited by-同舟云学术

Receptor for advanced glycation end products mediates sepsis-triggered amyloid-β accumulation, Tau phosphorylation, and cognitive impairment

Published:2018-01 Issue:1 Volume:293 Page:226-244
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Gasparotto Juciano,Girardi Carolina S.,Somensi Nauana,Ribeiro Camila T.,Moreira José C.F.,Michels Monique,Sonai Beatriz,Rocha Mariane,Steckert Amanda V.,Barichello Tatiana,Quevedo JoΔo,Dal-Pizzol Felipe,Gelain Daniel P.

Funder

Conselho Nacional de Desenvolvimento Científico e Tecnológico

Fundação de Amparo à Pesquisa do Estado do Rio Grande do Sul

Fundação de Amparo à Pesquisa e Inovação do Estado de Santa Catarina

Coordenação de Aperfeiçoamento de Pessoal de Nível Superior

University of Texas Medical School at Houston

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference83 articles.

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4. Sepsis causes neuroinflammation and concomitant decrease of cerebral metabolism;Semmler;J. Neuroinflammation,2008

5. Long-term mortality and quality of life in sepsis: a systematic review;Winters;Crit. Care Med,2010

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