STAT3β, a Splice Variant of Transcription Factor STAT3, Is a Dominant Negative Regulator of Transcription-Reference-Cited by-同舟云学术

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STAT3β, a Splice Variant of Transcription Factor STAT3, Is a Dominant Negative Regulator of Transcription

Published:1996-05 Issue:22 Volume:271 Page:13221-13227
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Caldenhoven Eric,van Dijk Thamar B.,Solari Roberto,Armstrong John,Raaijmakers Jan A.M.,Lammers Jan-Willem J.,Koenderman Leo,de Groot Rolf P.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference61 articles.

1. Protein kinases and phosphatases: The Yin and Yang of protein phosphorylation and signaling

2. Specificity of receptor tyrosine kinase signaling: Transient versus sustained extracellular signal-regulated kinase activation

3. Cytokine Signaling Through Nonreceptor Protein Tyrosine Kinases

4. Jaks and Stats in signaling by the cytokine receptor superfamily

5. ISGF3, the transcriptional activator induced by interferon alpha, consists of multiple interacting polypeptide chains.

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1. Identification of Entinostat as a Novel Modifier of STAT3 Pre-mRNA Alternative Splicing;Biological and Pharmaceutical Bulletin;2024-09-13

2. Aberrant alternative splicing in cancer: Splicing events and their regulatory mechanisms (Review);International Journal of Oncology;2024-08-02

3. Alternative Splicing of Pre-mRNA Matters in Oral Diseases;Current Gene Therapy;2024-07-23

4. A novel function of STAT3β in suppressing interferon response improves outcome in acute myeloid leukemia;Cell Death & Disease;2024-05-28

5. JAK/STAT3 signaling in cardiac fibrosis: a promising therapeutic target;Frontiers in Pharmacology;2024-03-01

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