Interleukin-1 Enhances Pancreatic Islet Arachidonic Acid 12-Lipoxygenase Product Generation by Increasing Substrate Availability through a Nitric Oxide-dependent Mechanism-Reference-Cited by-同舟云学术

Interleukin-1 Enhances Pancreatic Islet Arachidonic Acid 12-Lipoxygenase Product Generation by Increasing Substrate Availability through a Nitric Oxide-dependent Mechanism

Published:1996-01 Issue:2 Volume:271 Page:1029-1042
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Ma Zhongmin,Ramanadham Sasanka,Corbett John A.,Bohrer Alan,Gross Richard W.,McDaniel Michael L.,Turk John

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference89 articles.

1. Lipoxygenase Pathway in Islet Endocrine Cells. OXIDATIVE METABOLISM OF ARACHIDONIC ACID PROMOTES INSULIN RELEASE

2. Glucose increases the synthesis of lipoxygenase-mediated metabolites of arachidonic acid in intact rat islets.

3. Arachidonic acid metabolism in isolated pancreatic islets

4. Arachidonic acid metabolism in isolated pancreatic islets

5. Arachidonic acid metabolism in isolated pancreatic islets. III. Effects of exogenous lipoxygenase products and inhibitors on insulin secretion

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