The inhibition of cellular toxicity of amyloid-β by dissociated transthyretin
Author:
Funder
Amyloidosis Foundation
EC | Seventh Framework Programme
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference35 articles.
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2. β-Amyloid-(1–42) is a major component of cerebrovascular amyloid deposits: implications for the pathology of Alzheimer disease;Roher;Proc. Natl. Acad. Sci. U.S.A,1993
3. Assembly and aggregation properties of synthetic Alzheimer's A4/β amyloid peptide analogs;Burdick;J. Biol. Chem,1992
4. Neutralization of transthyretin reverses the neuroprotective effects of secreted amyloid precursor protein (APP) in APPSW mice resulting in tau phosphorylation and loss of hippocampal neurons: support for the amyloid hypothesis;Stein;J. Neurosci,2004
5. Accelerated Aβ deposition in APPswe/PS1deltaE9 mice with hemizygous deletions of TTR (transthyretin);Choi;J. Neurosci,2007
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