Agonist-independent Gαz activity negatively regulates beta-cell compensation in a diet-induced obesity model of type 2 diabetes-Reference-Cited by-同舟云学术

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Agonist-independent Gαz activity negatively regulates beta-cell compensation in a diet-induced obesity model of type 2 diabetes

Published:2021-01 Issue: Volume:296 Page:100056
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Schaid Michael D.^ORCID,Green Cara L.^ORCID,Peter Darby C.,Gallagher Shannon J.,Guthery Erin,Carbajal Kathryn A.,Harrington Jeffrey M.,Kelly Grant M.,Reuter Austin^ORCID,Wehner Molly L.,Brill Allison L.^ORCID,Neuman Joshua C.^ORCID,Lamming Dudley W.^ORCID,Kimple Michelle E.^ORCID

Funder

U.S. Department of Veterans Affairs

HHS | NIH | Office of Extramural Research, National Institutes of Health

American Society for Pharmacology and Experimental Therapeutics

American Federation for Aging Research

american diabetes association

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference50 articles.

1. Natural history of beta-cell adaptation and failure in type 2 diabetes;Alejandro;Mol. Aspects Med.,2015

2. cAMP signalling in insulin and glucagon secretion;Tengholm;Diabetes Obes. Metab.,2017

3. Medical treatment of diabetes mellitus;Skugor;Cleve Clin. J. Med.,2017

4. Inhibitory G proteins and their receptors: emerging therapeutic targets for obesity and diabetes;Kimple;Exp. Mol. Med.,2014

5. Synergy between galphaz deficiency and GLP-1 analog treatment in preserving functional beta-cell mass in experimental diabetes;Brill;Mol. Endocrinol.,2016

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