Co-aggregation of FcγRII with FcϵRI on Human Mast Cells Inhibits Antigen-induced Secretion and Involves SHIP-Grb2-Dok Complexes-Reference-Cited by-同舟云学术

Co-aggregation of FcγRII with FcϵRI on Human Mast Cells Inhibits Antigen-induced Secretion and Involves SHIP-Grb2-Dok Complexes

Published:2004-08 Issue:34 Volume:279 Page:35139-35149
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Kepley Christopher L.,Taghavi Sharven,Mackay Graham,Zhu Daocheng,Morel Penelope A.,Zhang Ke,Ryan John J.,Satin Leslie S.,Zhang Min,Pandolfi Pier P.,Saxon Andrew

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference64 articles.

1. Signalling through the high-affinity IgE receptor FcεRI

2. A 13-amino-acid motif in the cytoplasmic domain of FcγRIIB modulates B-cell receptor signalling

3. Recruitment and Activation of PTP1C in Negative Regulation of Antigen Receptor Signaling by FcγRIIB1

4. Regulation of high-affinity IgE receptor-mediated mast cell activation by murine low-affinity IgG receptors.

5. The same tyrosine-based inhibition motif, in the intra-cytoplasmic domain of FcγRIIB, regulates negatively BCR-, TCR-, and FcR-dependent cell activation

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