Overexpression of Phex in Osteoblasts Fails to Rescue the Hyp Mouse Phenotype
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference49 articles.
1. A gene (PEX) with homologies to endopeptidases is mutated in patients with X–linked hypophosphatemic rickets
2. Pex Gene Deletions in Gy and Hyp Mice Provide Mouse Models for X-Linked Hypophosphatemia
3. Expression and Cloning of the Human X-Linked Hypophosphatemia Gene cDNA
4. Endothelin-converting Enzyme-2 Is a Membrane-bound, Phosphoramidon-sensitive Metalloprotease with Acidic pH Optimum
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1. A multi-omics study to characterize the transdifferentiation of human dermal fibroblasts to osteoblast-like cells;Frontiers in Molecular Biosciences;2022-11-17
2. PHEXL222P Mutation Increases Phex Expression in a New ENU Mouse Model for XLH Disease;Genes;2022-07-28
3. Genetic Ablation of Osteopontin in Osteomalacic Hyp Mice Partially Rescues the Deficient Mineralization Without Correcting Hypophosphatemia;Journal of Bone and Mineral Research;2020-07-30
4. Molecular Control of Phosphorus Homeostasis and Precision Treatment of Hypophosphatemic Disorders;Current Molecular Biology Reports;2019-02-09
5. Heritable Renal Phosphate Wasting Disorders;Genetics of Bone Biology and Skeletal Disease;2018
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