Toll-like Receptor 4-mediated Endoplasmic Reticulum Stress in Intestinal Crypts Induces Necrotizing Enterocolitis-Reference-Cited by-同舟云学术

Toll-like Receptor 4-mediated Endoplasmic Reticulum Stress in Intestinal Crypts Induces Necrotizing Enterocolitis

Published:2014-04 Issue:14 Volume:289 Page:9584-9599
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Afrazi Amin,Branca Maria F.,Sodhi Chhinder P.,Good Misty,Yamaguchi Yukihiro,Egan Charlotte E.,Lu Peng,Jia Hongpeng,Shaffiey Shahab,Lin Joyce,Ma Congrong,Vincent Garrett,Thomas Prindle,Weyandt Samantha,Neal Matthew D.,Ozolek John A.,Wiersch John,Tschurtschenthaler Markus,Shiota Chiyo,Gittes George K.,Billiar Timothy R.,Mollen Kevin,Kaser Arthur,Blumberg Richard,Hackam David J.

Funder

National Institutes of Health

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference39 articles.

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2. Heparin-binding EGF-like growth factor protects intestinal stem cells from injury in a rat model of necrotizing enterocolitis;Chen;Lab. Invest.,2012

3. Toll-like receptor-4 inhibits enterocyte proliferation via impaired β-catenin signaling in necrotizing enterocolitis;Sodhi;Gastroenterology.,2010

4. Heparin-binding epidermal growth factor-like growth factor promotes enterocyte migration and proliferation in neonatal rats with necrotizing enterocolitis;Feng;J. Pediatr. Surg.,2007

5. A critical role for TLR4 in the pathogenesis of necrotizing enterocolitis by modulating intestinal injury and repair;Leaphart;J. Immunol.,2007

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