High Bone Mass in Mice Lacking Cx37 Because of Defective Osteoclast Differentiation-Reference-Cited by-同舟云学术

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High Bone Mass in Mice Lacking Cx37 Because of Defective Osteoclast Differentiation

Published:2014-03 Issue:12 Volume:289 Page:8508-8520
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Pacheco-Costa Rafael,Hassan Iraj,Reginato Rejane D.,Davis Hannah M.,Bruzzaniti Angela,Allen Matthew R.,Plotkin Lilian I.

Funder

National Institutes of Health

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference54 articles.

1. Beyond gap junctions: Connexin43 and bone cell signaling;Plotkin;Bone,2013

2. Gap junctions;Nielsen;Compr. Physiol,2012

3. Gap junctional channels are parts of multiprotein complexes;Hervé;Biochim. Biophys. Acta,2012

4. Regulation of connexin biosynthesis, assembly, gap junction formation, and removal;Segretain;Biochim. Biophys. Acta,2004

5. Functional analysis of selective interactions among rodent connexins;White;Mol. Biol. Cell,1995

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