Acetylcholine-induced Phosphatidylinositol 4,5-Bisphosphate Depletion Does Not Cause Short-term Desensitization of G Protein-gated Inwardly Rectifying K+ Current in Mouse Atrial Myocytes
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference32 articles.
1. Direct activation of inward rectifier potassium channels by PIP2 and its stabilization by Gβγ
2. Activation of inwardly rectifying K+ channels by distinct PtdIns(4,5)P2 interactions
3. Activation of the atrial KACh channel by the subunits of G proteins or intracellular Na+ ions depends on the presence of phosphatidylinositol phosphates
4. Molecular mechanism for sodium‐dependent activation of G protein‐gated K + channels
5. Gating of G protein‐sensitive inwardly rectifying K + channels through phosphatidylinositol 4,5‐bisphosphate
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