Passive Immunization against β-Amyloid Peptide Protects Central Nervous System (CNS) Neurons from Increased Vulnerability Associated with an Alzheimer’s Disease-causing Mutation
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference55 articles.
1. Formation of Neurofibrillary Tangles in P301L Tau Transgenic Mice Induced by Aβ42 Fibrils
2. Enhanced Neurofibrillary Degeneration in Transgenic Mice Expressing Mutant Tau and APP
3. Discussion
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1. Neuroprotective Immunity for Neurodegenerative and Neuroinfectious Diseases;Advanced Concepts in Human Immunology: Prospects for Disease Control;2020
2. Aging, Metabolism, Synaptic Activity, and Aβ in Alzheimer's Disease;Frontiers in Aging Neuroscience;2019-07-23
3. Kainate Receptor Activation Enhances Amyloidogenic Processing of APP in Astrocytes;Molecular Neurobiology;2018-11-27
4. A role for astrocyte‐derived amyloid β peptides in the degeneration of neurons in an animal model of temporal lobe epilepsy;Brain Pathology;2018-07-05
5. Lack of P-glycoprotein Results in Impairment of Removal of Beta-Amyloid and Increased Intraparenchymal Cerebral Amyloid Angiopathy after Active Immunization in a Transgenic Mouse Model of Alzheimer's Disease;Current Alzheimer Research;2017-05-09
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