Mutated Human SOD1 Causes Dysfunction of Oxidative Phosphorylation in Mitochondria of Transgenic Mice-Reference-Cited by-同舟云学术

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Mutated Human SOD1 Causes Dysfunction of Oxidative Phosphorylation in Mitochondria of Transgenic Mice

Published:2002-08 Issue:33 Volume:277 Page:29626-29633
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Mattiazzi Marina,D'Aurelio Marilena,Gajewski Carl D.,Martushova Katherine,Kiaei Mahmoud,Beal M. Flint,Manfredi Giovanni

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference40 articles.

1. Principles of Neurology;Adams,1997

2. Identification of two novel mutations and a new polymorphism in the gene for Cu/Zn superoxide dismutase in patients with amyotrophic lateral sclerosis

3. Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis

4. A frequent ala 4 to val superoxide dismutase-1 mutation is associated with a rapidly progressive familial amyotrophic lateral sclerosis

5. Rapid Communication: Cu/Zn Superoxide Dismutase Activity in Familial and Sporadic Amyotrophic Lateral Sclerosis

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