Molecular Mechanism and Functional Significance of the MinK Control of the KvLQT1 Channel Activity
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference28 articles.
1. Two components of cardiac delayed rectifier K+ current. Differential sensitivity to block by class III antiarrhythmic agents.
2. Positional cloning of a novel potassium channel gene: KVLQT1 mutations cause cardiac arrhythmias
3. KvLQT1 and IsK (minK) proteins associate to form the IKS cardiac potassium current
4. Coassembly of KVLQT1 and minK (IsK) proteins to form cardiac IKS potassium channel
5. Slowly activating voltage-dependent K+ conductance is apical pathway for K+ secretion in vestibular dark cells
Cited by 126 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Kv7 Channels and Excitability Disorders;Pharmacology of Potassium Channels;2021
2. Gating and Regulation of KCNQ1 and KCNQ1 + KCNE1 Channel Complexes;Frontiers in Physiology;2020-06-04
3. The calmodulin‐binding tetraleucine motif of KCNE4 is responsible for association with Kv1.3;The FASEB Journal;2019-04-10
4. Transcripts of Kv7.1 and MinK channels and slow delayed rectifier K+ current (IKs) are expressed in zebrafish (Danio rerio) heart;Pflügers Archiv - European Journal of Physiology;2018-08-16
5. D242N, a K V 7.1 LQTS mutation uncovers a key residue for I Ks voltage dependence;Journal of Molecular and Cellular Cardiology;2017-09
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线,采集、加工和组织学术论文而形成的新型学术文献查询和分析系统,可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容,当前同舟云学术共收录了国内外主流学术期刊6万余种,收集的期刊论文及会议论文总量共计约1.5亿篇,并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询!咨询电话:010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3