Transgenic Mice with Cardiac Overexpression of α1B-Adrenergic Receptors-Reference-Cited by-同舟云学术

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Transgenic Mice with Cardiac Overexpression of α1B-Adrenergic Receptors

Published:1997-08 Issue:34 Volume:272 Page:21253-21259
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Akhter Shahab A.,Milano Carmelo A.,Shotwell Kyle F.,Cho Myeong-Chan,Rockman Howard A.,Lefkowitz Robert J.,Koch Walter J.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference29 articles.

1. Protooncogene induction and reprogramming of cardiac gene expression produced by pressure overload.

2. Expression of a constitutively activated mutant of the beta-isozyme of protein kinase C in cardiac myocytes stimulates the promoter of the beta-myosin heavy chain isogene

3. Gq- and ras-dependent pathways mediate hypertrophy of neonatal rat ventricular myocytes following alpha 1-adrenergic stimulation.

4. Alpha- and beta-adrenergic stimulation induces distinct patterns of immediate early gene expression in neonatal rat myocardial cells. fos/jun expression is associated with sarcomere assembly; Egr-1 induction is primarily an alpha 1-mediated response.

5. Acquisition by innervated cardiac myocytes of a pertussis toxin-specific regulatory protein linked to the alpha 1-receptor

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