The JUN Kinase/Stress-activated Protein Kinase Pathway Is Required for Epidermal Growth Factor Stimulation of Growth of Human A549 Lung Carcinoma Cells-Reference-Cited by-同舟云学术

The JUN Kinase/Stress-activated Protein Kinase Pathway Is Required for Epidermal Growth Factor Stimulation of Growth of Human A549 Lung Carcinoma Cells

Published:1997-12 Issue:52 Volume:272 Page:33422-33429
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Bost Frédéric,McKay Robert,Dean Nicholas,Mercola Dan

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference76 articles.

1. Identification of an oncoprotein- and UV-responsive protein kinase that binds and potentiates the c-Jun activation domain.

2. The stress-activated protein kinase subfamily of c-Jun kinases

3. Dissection of TNF Receptor 1 Effector Functions: JNK Activation Is Not Linked to Apoptosis While NF-κB Activation Prevents Cell Death

4. The Jun Kinase/Stress-activated Protein Kinase Pathway Functions to Regulate DNA Repair and Inhibition of the Pathway Sensitizes Tumor Cells to Cisplatin

5. ATF-2 is preferentially activated by stress-activated protein kinases to mediate c-jun induction in response to genotoxic agents.

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