BH3 Domain of BAD Is Required for Heterodimerization with BCL-XL and Pro-apoptotic Activity
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference18 articles.
1. New members of the Bcl-2 family and their protein partners
2. Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell death
3. BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with Bax
4. Bax-independent inhibition of apoptosis by Bcl-XL
5. A conserved domain in Bak, distinct from BH1 and BH2, mediates cell death and protein binding functions.
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