Interleukin-1 (IL-1)-induced TAK1-dependent Versus MEKK3-dependent NFκB Activation Pathways Bifurcate at IL-1 Receptor-associated Kinase Modification-Reference-Cited by-同舟云学术

Interleukin-1 (IL-1)-induced TAK1-dependent Versus MEKK3-dependent NFκB Activation Pathways Bifurcate at IL-1 Receptor-associated Kinase Modification

Published:2007-03 Issue:9 Volume:282 Page:6075-6089
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Yao Jianhong,Kim Tae Whan,Qin Jinzhong,Jiang Zhengfan,Qian Youcun,Xiao Hui,Lu Yi,Qian Wen,Gulen Muhammet Fatih,Sizemore Nywana,DiDonato Joseph,Sato Shintaro,Akira Shizuo,Su Bing,Li Xiaoxia

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference50 articles.

1. Molecular Cloning and Characterization of a Second Subunit of the Interleukin 1 Receptor Complex

2. Recruitment of IRAK to the interleukin 1 receptor complex requires interleukin 1 receptor accessory protein

3. A critical role for interleukin-1 receptor accessory protein in interleukin-1 signaling

4. Targeted Disruption of the MyD88 Gene Results in Loss of IL-1- and IL-18-Mediated Function

5. MyD88: An Adapter That Recruits IRAK to the IL-1 Receptor Complex

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