Obesity Induces Hypothalamic Endoplasmic Reticulum Stress and Impairs Proopiomelanocortin (POMC) Post-translational Processing-Reference-Cited by-同舟云学术

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Obesity Induces Hypothalamic Endoplasmic Reticulum Stress and Impairs Proopiomelanocortin (POMC) Post-translational Processing

Published:2013-06 Issue:24 Volume:288 Page:17675-17688
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Çakir Isin,Cyr Nicole E.,Perello Mario,Litvinov Bogdan Patedakis,Romero Amparo,Stuart Ronald C.,Nillni Eduardo A.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference69 articles.

1. Studies on the physiological functions of the melanocortin system;Cone;Endocr. Rev,2006

2. Regulation of prohormone convertases in hypothalamic neurons. Implications for prothyrotropin-releasing hormone and proopiomelanocortin;Nillni;Endocrinology,2007

3. Regulation of the hypothalamic thyrotropin releasing hormone (TRH) neuron by neuronal and peripheral inputs;Nillni;Front. Neuroendocrinol,2010

4. Obesity and impaired prohormone processing associated with mutations in the human convertase 1 gene;Jackson;Nat. Genet,1997

5. A missense mutation disrupting a dibasic prohormone processing site in pro-opiomelanocortin (POMC) increases susceptibility to early-onset obesity through a novel molecular mechanism;Challis;Hum. Mol. Genet,2002

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