Transient Receptor Potential Channel 6 (TRPC6) Protects Podocytes during Complement-mediated Glomerular Disease-Reference-Cited by-同舟云学术

Transient Receptor Potential Channel 6 (TRPC6) Protects Podocytes during Complement-mediated Glomerular Disease

Published:2013-12 Issue:51 Volume:288 Page:36598-36609
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Kistler Andreas D.,Singh Geetika,Altintas Mehmet M.,Yu Hao,Fernandez Isabel C.,Gu Changkyu,Wilson Cory,Srivastava Sandeep Kumar,Dietrich Alexander,Walz Katherina,Kerjaschki Dontscho,Ruiz Phillip,Dryer Stuart,Sever Sanja,Dinda Amit K.,Faul Christian,Reiser Jochen

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference51 articles.

1. A mutation in the TRPC6 cation channel causes familial focal segmental glomerulosclerosis;Winn;Science,2005

2. TRPC6 is a glomerular slit diaphragm-associated channel required for normal renal function;Reiser;Nat. Genet,2005

3. A novel TRPC6 mutation that causes childhood FSGS;Heeringa;PLoS One,2009

4. Induction of TRPC6 channel in acquired forms of proteinuric kidney disease;Möller;J. Am. Soc. Nephrol,2007

5. Sensitizing the slit diaphragm with TRPC6 ion channels;Möller;J. Am. Soc. Nephrol,2009

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