The ABCG5 ABCG8 Sterol Transporter Opposes the Development of Fatty Liver Disease and Loss of Glycemic Control Independently of Phytosterol Accumulation-Reference-Cited by-同舟云学术

The ABCG5 ABCG8 Sterol Transporter Opposes the Development of Fatty Liver Disease and Loss of Glycemic Control Independently of Phytosterol Accumulation

Published:2012-08 Issue:34 Volume:287 Page:28564-28575
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Su Kai,Sabeva Nadezhda S.,Liu Jingjing,Wang Yuhuan,Bhatnagar Saloni,van der Westhuyzen Deneys R.,Graf Gregory A.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference41 articles.

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2. Overexpression of ABCG5 and ABCG8 promotes biliary cholesterol secretion and reduces fractional absorption of dietary cholesterol;Yu;J. Clin. Investig.,2002

3. ABCG5 and ABCG8 are obligate heterodimers for protein trafficking and biliary cholesterol excretion;Graf;J. Biol. Chem.,2003

4. Sitosterolemia;Salen;J. Lipid Res.,1992

5. Accumulation of dietary cholesterol in sitosterolemia caused by mutations in adjacent ABC transporters;Berge;Science,2000

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