M8R tropomyosin mutation disrupts actin binding and filament regulation: The beginning affects the middle and end
Author:
Funder
American Heart Association
HHS | NIH | National Heart, Lung, and Blood Institute
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference47 articles.
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3. Targets for therapy in sarcomeric cardiomyopathies;Tardiff;Cardiovasc. Res,2015
4. HCM and DCM cardiomyopathy-linked α-tropomyosin mutations influence off-state stability and crossbridge interaction on thin filaments;Farman;Arch. Biochem. Biophys,2018
5. Cardiomyopathy mutation alters end-to-end junction of tropomyosin and reduces calcium sensitivity;Sundar;Biophys. J,2020
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3. Alternative splicing of a single exon causes a major impact on the affinity of Caenorhabditis elegans tropomyosin isoforms for actin filaments;Frontiers in Cell and Developmental Biology;2023-09-07
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