Human Serum from Patients with Septic Shock Activates Transcription Factors STAT1, IRF1, and NF-κB and Induces Apoptosis in Human Cardiac Myocytes-Reference-Cited by-同舟云学术

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Human Serum from Patients with Septic Shock Activates Transcription Factors STAT1, IRF1, and NF-κB and Induces Apoptosis in Human Cardiac Myocytes

Published:2005-12 Issue:52 Volume:280 Page:42619-42626
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Kumar Aseem,Kumar Anand,Michael Paul,Brabant Danielle,Parissenti Amadeo M.,Ramana Chilakamarti V.,Xu Xiulong,Parrillo Joseph E.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference75 articles.

1. Profound but Reversible Myocardial Depression in Patients with Septic Shock

2. Pathogenetic Mechanisms of Septic Shock

3. A circulating myocardial depressant substance in humans with septic shock. Septic shock patients with a reduced ejection fraction have a circulating factor that depresses in vitro myocardial cell performance.

4. Tumor necrosis factor alpha and interleukin 1beta are responsible for in vitro myocardial cell depression induced by human septic shock serum.

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3. KLF13 overexpression protects sepsis‐induced myocardial injury and LPS ‐induced inflammation and apoptosis;International Journal of Experimental Pathology;2022-12-30

4. Triiodothyronine enhances cardiac contractility in septic rats and probably through Akt-Caspase9 pathway to reduce septic-induced cardiomyocyte apoptosis;Molecular and Cellular Probes;2022-12

5. Identification of the TF-miRNA-mRNA co-regulatory networks involved in sepsis;Functional & Integrative Genomics;2022-03-24

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