HSCARG Regulates NF-κB Activation by Promoting the Ubiquitination of RelA or COMMD1
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference45 articles.
1. Death and NF-κB in T Cell Activation: Life at the Edge
2. NF-κB at the crossroads of life and death
3. The Rel/NF-κB family: friend and foe
4. COMMD Proteins and the Control of the NFκB Pathway
5. Signal-induced site-specific phosphorylation targets I kappa B alpha to the ubiquitin-proteasome pathway.
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2. Clinical Significance of HSCARG for Atherosclerotic Coronary Heart Disease and Reduced ROS-Oxidative Stress in in Vivo and in Vitro Models via p47phox by NF-κB Activity;Brazilian Journal of Cardiovascular Surgery;2022
3. Structure and functions of cellular redox sensor HSCARG/NMRAL1, a linkage among redox status, innate immunity, DNA damage response, and cancer;Free Radical Biology and Medicine;2020-11
4. MTSS1 suppresses mammary tumor-initiating cells by enhancing RBCK1-mediated p65 ubiquitination;Nature Cancer;2020-01-20
5. Identification of COMMD1 as a novel lamin A binding partner;Molecular Medicine Reports;2019-06-24
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