TDAG51 Is Induced by Homocysteine, Promotes Detachment-mediated Programmed Cell Death, and Contributes to the Development of Atherosclerosis in Hyperhomocysteinemia-Reference-Cited by-同舟云学术

TDAG51 Is Induced by Homocysteine, Promotes Detachment-mediated Programmed Cell Death, and Contributes to the Development of Atherosclerosis in Hyperhomocysteinemia

Published:2003-08 Issue:32 Volume:278 Page:30317-30327
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Hossain Gazi S.,van Thienen Johannes V.,Werstuck Geoff H.,Zhou Ji,Sood Sudesh K.,Dickhout Jeffrey G.,de Koning A.B.Lawrence,Tang Damu,Wu Dongcheng,Falk Erling,Poddar Ranjana,Jacobsen Donald W.,Zhang Kezhong,Kaufman Randal J.,Austin Richard C.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference50 articles.

1. A Quantitative Assessment of Plasma Homocysteine as a Risk Factor for Vascular Disease

2. Homocyst(e)ine and Cardiovascular Disease: A Critical Review of the Epidemiologic Evidence

3. Homocyst(e)ine, Diet, and Cardiovascular Diseases

4. Homocysteine and Atherothrombosis

5. Hyperhomocysteinemia enhances vascular inflammation and accelerates atherosclerosis in a murine model

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