Elimination of KATP Channels in Mouse Islets Results in Elevated [U-13C]Glucose Metabolism, Glutaminolysis, and Pyruvate Cycling but a Decreased γ-Aminobutyric Acid Shunt-Reference-Cited by-同舟云学术

Elimination of KATP Channels in Mouse Islets Results in Elevated [U-13C]Glucose Metabolism, Glutaminolysis, and Pyruvate Cycling but a Decreased γ-Aminobutyric Acid Shunt

Published:2008-06 Issue:25 Volume:283 Page:17238-17249
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Li Changhong,Nissim Itzhak,Chen Pan,Buettger Carol,Najafi Habiba,Daikhin Yevgeny,Nissim Ilana,Collins Heather W.,Yudkoff Marc,Stanley Charles A.,Matschinsky Franz M.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference45 articles.

1. The Network of Glucokinase-Expressing Cells in Glucose Homeostasis and the Potential of Glucokinase Activators for Diabetes Therapy

2. Triggering and amplifying pathways of regulation of insulin secretion by glucose.

3. Stimulus-secretion coupling in the pancreatic B-cell: The cholinergic pathway for insulin release

4. Restitution of defective glucose-stimulated insulin release of sulfonylurea type 1 receptor knockout mice by acetylcholine

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