Tumor Necrosis Factor-α Activation of the c-Jun N-terminal Kinase Pathway in Human Neutrophils-Reference-Cited by-同舟云学术

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Tumor Necrosis Factor-α Activation of the c-Jun N-terminal Kinase Pathway in Human Neutrophils

Published:2001-01 Issue:3 Volume:276 Page:2189-2199
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Avdi Natalie J.,Nick Jerry A.,Whitlock Ben B.,Billstrom Marcella A.,Henson Peter M.,Johnson Gary L.,Worthen G. Scott

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference95 articles.

1. Human Inflammatory Disease, Clinical Immunology;Henson,1988

2. Neutrophils, host defense, and inflammation: a double-edged sword

3. Endotoxin-responsive sequences control cachectin/tumor necrosis factor biosynthesis at the translational level.

4. Tumor necrosis factor-alpha blockade prevents neutrophil CD18 receptor upregulation and attenuates acute lung injury in porcine sepsis without inhibition of neutrophil oxygen radical generation.

5. Delayed Tumor Necrosis Factor α Blockade Attenuates Pulmonary Dysfunction and Metabolic Acidosis Associated With Experimental Gram-negative Sepsis

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