An Arg307 to Gln Polymorphism within the ATP-binding Site Causes Loss of Function of the Human P2X7 Receptor-Reference-Cited by-同舟云学术

An Arg307 to Gln Polymorphism within the ATP-binding Site Causes Loss of Function of the Human P2X7 Receptor

Published:2004-07 Issue:30 Volume:279 Page:31287-31295
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Gu Ben J.,Sluyter Ronald,Skarratt Kristen K.,Shemon Anne N.,Dao-Ung Lan-Phuong,Fuller Stephen J.,Barden Julian A.,Clarke Alison L.,Petrou Steven,Wiley James S.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference43 articles.

1. Stress-activated Protein Kinase/JNK Activation and Apoptotic Induction by the Macrophage P2X7 Nucleotide Receptor

2. P2Z/P2X7receptor-dependent apoptosis of dendritic cells

3. Nucleotide receptors: an emerging family of regulatory molecules in blood cells

4. Molecular Physiology of P2X Receptors

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