Mrs3p, Mrs4p, and Frataxin Provide Iron for Fe-S Cluster Synthesis in Mitochondria
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference42 articles.
1. Friedreich's Ataxia: Autosomal Recessive Disease Caused by an Intronic GAA Triplet Repeat Expansion
2. Mouse models for Friedreich ataxia exhibit cardiomyopathy, sensory nerve defect and Fe-S enzyme deficiency followed by intramitochondrial iron deposits
3. Regulation of Mitochondrial Iron Accumulation by Yfh1p, a Putative Homolog of Frataxin
4. Aconitase and mitochondrial iron–sulphur protein deficiency in Friedreich ataxia
5. Iron use for haeme synthesis is under control of the yeast frataxin homologue (Yfh1)
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