Peroxisome Proliferator-activated Receptor γ Coactivator 1α or 1β Overexpression Inhibits Muscle Protein Degradation, Induction of Ubiquitin Ligases, and Disuse Atrophy
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference71 articles.
1. Muscle Protein Breakdown and the Critical Role of the Ubiquitin-Proteasome Pathway in Normal and Disease States
2. Inhibitors of the proteasome reduce the accelerated proteolysis in atrophying rat skeletal muscles.
3. Multiple types of skeletal muscle atrophy involve a common program of changes in gene expression
4. Rapid disuse and denervation atrophy involve transcriptional changes similar to those of muscle wasting during systemic diseases
5. Foxo Transcription Factors Induce the Atrophy-Related Ubiquitin Ligase Atrogin-1 and Cause Skeletal Muscle Atrophy
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