Multiple Members of the Mitogen-activated Protein Kinase Family Are Necessary for PED/PEA-15 Anti-apoptotic Function-Reference-Cited by-同舟云学术

Multiple Members of the Mitogen-activated Protein Kinase Family Are Necessary for PED/PEA-15 Anti-apoptotic Function

Published:2002-03 Issue:13 Volume:277 Page:11013-11018
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Condorelli Gerolama,Trencia Alessandra,Vigliotta Giovanni,Perfetti Anna,Goglia Umberto,Cassese Angela,Musti Anna Maria,Miele Claudia,Santopietro Stefania,Formisano Pietro,Beguinot Francesco

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference28 articles.

1. Characterization of PEA-15, a major substrate for protein kinase C in astrocytes.

2. PED/PEA-15 gene controls glucose transport and is overexpressed in type 2 diabetes mellitus

3. PED/PEA-15: an anti-apoptotic molecule that regulates FAS/TNFR1-induced apoptosis

4. FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling Complex

5. Knock-Out of the Neural Death Effector Domain Protein PEA-15 Demonstrates That Its Expression Protects Astrocytes from TNFα-Induced Apoptosis

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2. Phosphoprotein enriched in diabetes (PED/PEA15) promotes migration in hepatocellular carcinoma and confers resistance to sorafenib;Cell Death & Disease;2017-10

3. Targetting PED/PEA-15 for diabetes treatment;Expert Opinion on Therapeutic Targets;2017-04-21

4. Integrin α5β1 and p53 convergent pathways in the control of anti-apoptotic proteins PEA-15 and survivin in high-grade glioma;Cell Death & Differentiation;2015-10-16

5. PED/PEA-15 Inhibits Hydrogen Peroxide-Induced Apoptosis in Ins-1E Pancreatic Beta-Cells via PLD-1;PLoS ONE;2014-12-09