Calpain Activation Promotes BACE1 Expression, Amyloid Precursor Protein Processing, and Amyloid Plaque Formation in a Transgenic Mouse Model of Alzheimer Disease-Reference-Cited by-同舟云学术

Calpain Activation Promotes BACE1 Expression, Amyloid Precursor Protein Processing, and Amyloid Plaque Formation in a Transgenic Mouse Model of Alzheimer Disease

Published:2010-09 Issue:36 Volume:285 Page:27737-27744
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Liang Bin,Duan Bao-Yu,Zhou Xiu-Ping,Gong Jia-Xin,Luo Zhen-Ge

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference67 articles.

1. The Amyloid Hypothesis of Alzheimer's Disease: Progress and Problems on the Road to Therapeutics

2. Soluble protein oligomers in neurodegeneration: lessons from the Alzheimer's amyloid β-peptide

3. Cortical Synaptic Integration In Vivo Is Disrupted by Amyloid- Plaques

4. Plaque-induced neurite abnormalities: Implications for disruption of neural networks in Alzheimer's disease

5. In Vivo Multiphoton Imaging of a Transgenic Mouse Model of Alzheimer Disease Reveals Marked Thioflavine-S-Associated Alterations in Neurite Trajectories

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