Ca2+ Overload and Sarcoplasmic Reticulum Instability in tric-a Null Skeletal Muscle-Reference-Cited by-同舟云学术

Ca2+ Overload and Sarcoplasmic Reticulum Instability in tric-a Null Skeletal Muscle

Published:2010-11 Issue:48 Volume:285 Page:37370-37376
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Zhao Xiaoli,Yamazaki Daiju,Park Ki Ho,Komazaki Shinji,Tjondrokoesoemo Andoria,Nishi Miyuki,Lin Peihui,Hirata Yutaka,Brotto Marco,Takeshima Hiroshi,Ma Jianjie

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference26 articles.

1. The mechanical hypothesis of excitation—contraction (EC) coupling in skeletal muscle

2. The role of Ca2+ ions in excitation-contraction coupling of skeletal muscle fibres

3. Ryanodine Receptor Calcium Release Channels

4. Uncoupling Store-Operated Ca2+ Entry and Altered Ca2+ Release from Sarcoplasmic Reticulum through Silencing of Junctophilin Genes

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