Chronic and Acute Models of Retinal Neurodegeneration TrkA Activity Are Neuroprotective whereas p75NTR Activity Is Neurotoxic through a Paracrine Mechanism-Reference-Cited by-同舟云学术

Chronic and Acute Models of Retinal Neurodegeneration TrkA Activity Are Neuroprotective whereas p75NTR Activity Is Neurotoxic through a Paracrine Mechanism

Published:2010-12 Issue:50 Volume:285 Page:39392-39400
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Bai Yujing,Dergham Pauline,Nedev Hinyu,Xu Jing,Galan Alba,Rivera Jose Carlos,ZhiHua Shi,Mehta Hrishikesh M.,Woo Sang B.,Sarunic Marinko V.,Neet Kenneth E.,Saragovi H. Uri

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference36 articles.

1. New paradigms in the mechanisms and management of glaucoma

2. A Neurotrophic Rationale for the Therapy of Neurodegenerative Disorders

3. Neurotrophic rationale in glaucoma: A TrkA agonist, but not NGF or a p75 antagonist, protects retinal ganglion cellsin vivo

4. An Agonistic TrkB mAb Causes Sustained TrkB Activation, Delays RGC Death, and Protects the Retinal Structure in Optic Nerve Axotomy and in Glaucoma

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3. Neurotrophins and Trk Neurotrophin Receptors in the Retina of Adult Killifish (Nothobranchius guentheri);International Journal of Molecular Sciences;2024-02-27

4. Differential contribution of TrkB and p75NTR to BDNF-dependent self-renewal, proliferation, and differentiation of adult neural stem cells;Frontiers in Molecular Neuroscience;2023-12-22

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