Cardiac Myocyte-specific Ablation of Follistatin-like 3 Attenuates Stress-induced Myocardial Hypertrophy-Reference-Cited by-同舟云学术

Cardiac Myocyte-specific Ablation of Follistatin-like 3 Attenuates Stress-induced Myocardial Hypertrophy

Published:2011-03 Issue:11 Volume:286 Page:9840-9848
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Shimano Masayuki,Ouchi Noriyuki,Nakamura Kazuto,Oshima Yuichi,Higuchi Akiko,Pimentel David R.,Panse Kalyani D.,Lara-Pezzi Enrique,Lee Se-Jin,Sam Flora,Walsh Kenneth

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference36 articles.

1. Prognostic Implications of Echocardiographically Determined Left Ventricular Mass in the Framingham Heart Study

2. Cytoplasmic Signaling Pathways That Regulate Cardiac Hypertrophy

3. A Secretion Trap Screen in Yeast Identifies Protease Inhibitor 16 as a Novel Antihypertrophic Protein Secreted From the Heart

4. Adiponectin-mediated modulation of hypertrophic signals in the heart

5. Disruption of coordinated cardiac hypertrophy and angiogenesis contributes to the transition to heart failure

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