α1,6-Fucosyltransferase-deficient Mice Exhibit Multiple Behavioral Abnormalities Associated with a Schizophrenia-like Phenotype-Reference-Cited by-同舟云学术

α1,6-Fucosyltransferase-deficient Mice Exhibit Multiple Behavioral Abnormalities Associated with a Schizophrenia-like Phenotype

Published:2011-05 Issue:21 Volume:286 Page:18434-18443
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Fukuda Tomohiko,Hashimoto Hirokazu,Okayasu Natsumi,Kameyama Akihiko,Onogi Hiroshi,Nakagawasai Osamu,Nakazawa Takahiro,Kurosawa Tomoyo,Hao Yan,Isaji Tomoya,Tadano Takeshi,Narimatsu Hisashi,Taniguchi Naoyuki,Gu Jianguo

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference64 articles.

1. The control of glycoprotein synthesis: N-acetylgluocosamine linkage to a mannose residue as a signal for the attachment of L-fucose to the asparagine-linked N-acetylglucosamine residue of glycopeptide from α1-acid glycoprotein

2. Purification and cDNA Cloning of Porcine Brain GDP-L-Fuc:N-Acetyl-β-D-Glucosaminide α1→6Fucosyltransferase

3. Structures of N-Linked Sugar Chains Expressed Mainly in Mouse Brain

4. From The Cover: Dysregulation of TGF- 1 receptor activation leads to abnormal lung development and emphysema-like phenotype in core fucose-deficient mice

5. Requirement of Fut8 for the expression of vascular endothelial growth factor receptor-2: a new mechanism for the emphysema-like changes observed in Fut8-deficient mice

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