Deceleration of the E1P-E2P Transition and Ion Transport by Mutation of Potentially Salt Bridge-forming Residues Lys-791 and Glu-820 in Gastric H+/K+-ATPase
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference60 articles.
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1. Specific protonation of acidic residues confers K+ selectivity to the gastric proton pump;Journal of Biological Chemistry;2024-01
2. Gastric proton pump with two occluded K+ engineered with sodium pump-mimetic mutations;Nature Communications;2021-09-29
3. Gastric H+, K+-ATPase inhibitory effects of the active constituent isolated from Potentilla fulgens roots: An in vivo and in silico molecular docking studies;Phytomedicine Plus;2021-08
4. Displacement of the Na+/K+ pump’s transmembrane domains demonstrates conserved conformational changes in P-type 2 ATPases;Proceedings of the National Academy of Sciences;2021-02-17
5. K+ binding and proton redistribution in the E2P state of the H+, K+-ATPase;Scientific Reports;2018-08-24
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