Phosphorylation of STAT-3 in Response to Basic Fibroblast Growth Factor Occurs through a Mechanism Involving Platelet-activating Factor, JAK-2, and Src in Human Umbilical Vein Endothelial Cells-Reference-Cited by-同舟云学术

Phosphorylation of STAT-3 in Response to Basic Fibroblast Growth Factor Occurs through a Mechanism Involving Platelet-activating Factor, JAK-2, and Src in Human Umbilical Vein Endothelial Cells

Published:2002-06 Issue:24 Volume:277 Page:21237-21245
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Deo Dayanand D.,Axelrad T. William,Robert Everett G.,Marcheselli Victor,Bazan Nicolas G.,Hunt Jay D.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference56 articles.

1. Platelet-Activating Factor and Related Lipid Mediators

2. Thrombopoietin Stimulates Endothelial Cell Motility and Neoangiogenesis by a Platelet-Activating Factor–Dependent Mechanism

3. Potential Angiogenic Role of Platelet-Activating Factor in Human Breast Cancer

4. Tumor necrosis factor alpha-induced angiogenesis depends on in situ platelet-activating factor biosynthesis.

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