Adaptations of Energy Metabolism Associated with Increased Levels of Mitochondrial Cholesterol in Niemann-Pick Type C1-deficient Cells
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference56 articles.
1. Identification of surface residues on Niemann-Pick C2 essential for hydrophobic handoff of cholesterol to NPC1 in lysosomes;Wang;Cell Metab,2010
2. Niemann-Pick type C1 function requires lumenal domain residues that mediate cholesterol-dependent NPC2 binding;Deffieu;Proc. Natl. Acad. Sci. U.S.A,2011
3. NPC2 facilitates bidirectional transfer of cholesterol between NPC1 and lipid bilayers, a step in cholesterol egress from lysosomes;Infante;Proc. Natl. Acad. Sci. U.S.A,2008
4. Altered cholesterol metabolism in Niemann-Pick type C1 mouse brains affects mitochondrial function;Yu;J. Biol. Chem,2005
5. MLN64 mediates egress of cholesterol from endosomes to mitochondria in the absence of functional Niemann-Pick Type C1 protein;Charman;J. Lipid Res,2010
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