Defects in the Leptin Axis Reduce Abundance of the ABCG5-ABCG8 Sterol Transporter in Liver
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference51 articles.
1. Accumulation of Dietary Cholesterol in Sitosterolemia Caused by Mutations in Adjacent ABC Transporters
2. Identification of a gene, ABCG5, important in the regulation of dietary cholesterol absorption
3. Disruption of Abcg5 and Abcg8 in mice reveals their crucial role in biliary cholesterol secretion
4. A mouse model of sitosterolemia: absence of Abcg8/sterolin-2 results in failure to secrete biliary cholesterol
5. Sitosterolemia in ABC-Transporter G5-deficient mice is aggravated on activation of the liver-X receptor
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