Suppression of p38 MAPK and JNK via Akt-mediated Inhibition of Apoptosis Signal-regulating Kinase 1 Constitutes a Core Component of the β-Cell Pro-survival Effects of Glucose-dependent Insulinotropic Polypeptide-Reference-Cited by-同舟云学术

Suppression of p38 MAPK and JNK via Akt-mediated Inhibition of Apoptosis Signal-regulating Kinase 1 Constitutes a Core Component of the β-Cell Pro-survival Effects of Glucose-dependent Insulinotropic Polypeptide

Published:2009-10 Issue:44 Volume:284 Page:30372-30382
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Widenmaier Scott B.,Ao Ziliang,Kim Su-Jin,Warnock Garth,McIntosh Christopher H.S.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference65 articles.

1. Etiology of Insulin Resistance

2. Pathogenesis of Type 2 Diabetes Mellitus

3. Five Stages of Evolving Beta-Cell Dysfunction During Progression to Diabetes

4. The relative contributions of insulin resistance and beta-cell dysfunction to the pathophysiology of Type 2 diabetes

5. Type 2 Diabetes-a Matter of ß-Cell Life and Death?

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