Activating Transcription Factor 3 Activates p53 by Preventing E6-associated Protein from Binding to E6
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference58 articles.
1. Papillomaviruses Causing Cancer: Evasion From Host-Cell Control in Early Events in Carcinogenesis
2. Human papillomavirus and cervical cancer
3. The E6 oncoprotein encoded by human papillomavirus types 16 and 18 promotes the degradation of p53
4. High-risk human papillomavirus E6 protein has two distinct binding sites within p53, of which only one determines degradation
5. A cellular protein mediates association of p53 with the E6 oncoprotein of human papillomavirus types 16 or 18.
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1. Activating transcription factor 3 mediates apoptosis and cell cycle arrest in TP53-mutated anaplastic thyroid cancer cells;Thyroid Research;2024-08-01
2. The Dual Roles of Activating Transcription Factor 3 (ATF3) in Inflammation, Apoptosis, Ferroptosis, and Pathogen Infection Responses;International Journal of Molecular Sciences;2024-01-09
3. Deregulation of host gene expression by HPV16 E8^E2 knock-out genomes is due to increased productive replication;Virology;2023-04
4. Activating transcription factor 3 inhibits NF‑κB p65 signaling pathway and mediates apoptosis and cell cycle arrest in cervical cancer cells;Infectious Agents and Cancer;2022-12-15
5. A small-molecule inhibitor of MDMX suppresses cervical cancer cells via the inhibition of E6-E6AP-p53 axis;Pharmacological Research;2022-03
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