Phosphorylation of Human Rad9 Is Required for Genotoxin-activated Checkpoint Signaling-Reference-Cited by-同舟云学术

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Phosphorylation of Human Rad9 Is Required for Genotoxin-activated Checkpoint Signaling

Published:2003-07 Issue:27 Volume:278 Page:24428-24437
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Roos-Mattjus Pia,Hopkins Kevin M.,Oestreich Andrea J.,Vroman Benjamin T.,Johnson Kenneth L.,Naylor Stephen,Lieberman Howard B.,Karnitz Larry M.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference59 articles.

1. Sensing and responding to DNA damage

2. The DNA damage response: putting checkpoints in perspective

3. A unified view of the DNA-damage checkpoint

4. Mammalian G1- and S-phase checkpoints in response to DNA damage

5. The G2-phase DNA-damage checkpoint

Cited by 106 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Structural basis for intra- and intermolecular interactions on RAD9 subunit of 9-1-1 checkpoint clamp implies functional 9-1-1 regulation by RHINO;Journal of Biological Chemistry;2024-03

2. Resection of DNA double-strand breaks activates Mre11–Rad50–Nbs1- and Rad9–Hus1–Rad1-dependent mechanisms that redundantly promote ATR checkpoint activation and end processing in Xenopus egg extracts;Nucleic Acids Research;2024-02-13

3. Unique and shared functions of the Rad9–Hus1–Rad1 and Mre11–Rad50–Nbs1 complexes in ATR checkpoint activation and long-range DNA end resection inXenopusegg extracts;2023-08-10

4. DNA binding by the Rad9A subunit of the Rad9-Rad1-Hus1 complex;PLOS ONE;2022-08-08

5. Exonucleases: Degrading DNA to Deal with Genome Damage, Cell Death, Inflammation and Cancer;Cells;2022-07-09

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