Loss of Cleavage at β′-Site Contributes to Apparent Increase in β-Amyloid Peptide (Aβ) Secretion by β-Secretase (BACE1)-Glycosylphosphatidylinositol (GPI) Processing of Amyloid Precursor Protein-Reference-Cited by-同舟云学术

Loss of Cleavage at β′-Site Contributes to Apparent Increase in β-Amyloid Peptide (Aβ) Secretion by β-Secretase (BACE1)-Glycosylphosphatidylinositol (GPI) Processing of Amyloid Precursor Protein

Published:2011-07 Issue:29 Volume:286 Page:26166-26177
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Vetrivel Kulandaivelu S.,Barman Arghya,Chen Ying,Nguyen Phuong D.,Wagner Steven L.,Prabhakar Rajeev,Thinakaran Gopal

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference46 articles.

1. Amyloid Precursor Protein Trafficking, Processing, and Function

2. β-Secretase Cleavage of Alzheimer's Amyloid Precursor Protein by the Transmembrane Aspartic Protease BACE

3. Purification and cloning of amyloid precursor protein β-secretase from human brain

4. Membrane-anchored aspartyl protease with Alzheimer's disease β-secretase activity

5. BACE1 is the major β-secretase for generation of Aβ peptides by neurons

Cited by 31 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Cholesterol and Lipid Rafts in the Biogenesis of Amyloid-β Protein and Alzheimer's Disease;Annual Review of Biophysics;2024-07-16

2. Unraveling the role of miRNAs in the diagnosis, progression, and therapeutic intervention of Alzheimer’s disease;Pathology - Research and Practice;2024-01

3. Lost in traffic: consequences of altered palmitoylation in neurodegeneration;Frontiers in Physiology;2023-05-30

4. Lipid flippase dysfunction as a therapeutic target for endosomal anomalies in Alzheimer’s disease;iScience;2022-03

5. Lipid flippase dysfunction as a novel therapeutic target for endosomal anomalies in Alzheimer’s disease;2021-07-30