Selenoprotein K Is a Novel Target of m-Calpain, and Cleavage Is Regulated by Toll-like Receptor-induced Calpastatin in Macrophages
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference36 articles.
1. Calpain Expression in Lymphoid Cells
2. Regulation of STAT signalling by proteolytic processing
3. Calpain 2 Controls Turnover of LFA-1 Adhesions on Migrating T Lymphocytes
4. Structural modifications associated with the change in Ca2+sensitivity on activation of m-calpain
5. The crystal structure of calcium-free human m-calpain suggests an electrostatic switch mechanism for activation by calcium
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