Glucose-dependent Insulinotropic Polypeptide (GIP) Stimulation of Pancreatic β-Cell Survival Is Dependent upon Phosphatidylinositol 3-Kinase (PI3K)/Protein Kinase B (PKB) Signaling, Inactivation of the Forkhead Transcription Factor Foxo1, and Down-regulation of bax Expression-Reference-Cited by-同舟云学术

Glucose-dependent Insulinotropic Polypeptide (GIP) Stimulation of Pancreatic β-Cell Survival Is Dependent upon Phosphatidylinositol 3-Kinase (PI3K)/Protein Kinase B (PKB) Signaling, Inactivation of the Forkhead Transcription Factor Foxo1, and Down-regulation of bax Expression

Published:2005-06 Issue:23 Volume:280 Page:22297-22307
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Kim Su-Jin,Winter Kyle,Nian Cuilan,Tsuneoka Makoto,Koda Yoshiro,McIntosh Christopher H.S.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference41 articles.

1. Transcription-dependent and -independent control of neuronal survival by the PI3K–Akt signaling pathway

2. PI3K: Downstream AKTion Blocks Apoptosis

3. Regulation of Cell Death Protease Caspase-9 by Phosphorylation

4. Akt Phosphorylation of BAD Couples Survival Signals to the Cell-Intrinsic Death Machinery

5. Role of Glycogen Synthase Kinase-3 in the Phosphatidylinositol 3-Kinase/Akt Cell Survival Pathway

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