The p75 Neurotrophin Receptor (p75NTR) Alters Tumor Necrosis Factor-mediated NF-κB Activity under Physiological Conditions, but Direct p75NTR-mediated NF-κB Activation Requires Cell Stress-Reference-Cited by-同舟云学术

The p75 Neurotrophin Receptor (p75NTR) Alters Tumor Necrosis Factor-mediated NF-κB Activity under Physiological Conditions, but Direct p75NTR-mediated NF-κB Activation Requires Cell Stress

Published:1999-07 Issue:30 Volume:274 Page:21443-21449
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Bhakar Asha L.,Roux Philippe P.,Lachance Christian,Kryl David,Zeindler Christine,Barker Philip A.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference50 articles.

1. Activation of MAP kinase kinase is necessary and sufficient for PC12 differentiation and for transformation of NIH 3T3 cells

2. Mice lacking brain-derived neurotrophic factor develop with sensory deficits

3. Lack of neurotrophin-3 leads to deficiencies in the peripheral nervous system and loss of limb proprioceptive afferents

4. Targeted disruption of the BDNF gene perturbs brain and sensory neuron development but not motor neuron development

5. Targeted disruption of the trkB neurotrophin receptor gene results in nervous system lesions and neonatal death

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