Early Phenotypic Changes in Transgenic Mice That Overexpress Different Mutants of Amyloid Precursor Protein in Brain-Reference-Cited by-同舟云学术

Early Phenotypic Changes in Transgenic Mice That Overexpress Different Mutants of Amyloid Precursor Protein in Brain

Published:1999-03 Issue:10 Volume:274 Page:6483-6492
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Moechars Dieder,Dewachter Ilse,Lorent Kristin,Reversé Delphine,Baekelandt Veerle,Naidu Asha,Tesseur Ina,Spittaels Kurt,Haute Chris Van Den,Checler Fréderic,Godaux Emile,Cordell Barbara,Van Leuven Fred

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference38 articles.

1. Amyloid β-Protein and the Genetics of Alzheimer's Disease

2. A pathogenic mutation for probable Alzheimer's disease in the APP gene at the N–terminus of β–amyloid

3. Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's disease

4. Evidence that the 42- and 40-amino acid forms of amyloid protein are generated from the -amyloid precursor protein by different protease activities

5. Evidence that A?42 is the real culprit in alzheimer's disease

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