Functional Consequences of the Sustained or Transient Activation by Bax of the Mitochondrial Permeability Transition Pore
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference34 articles.
1. The Overexpression of Bax Produces Cell Death upon Induction of the Mitochondrial Permeability Transition
2. Increase in bax expression in substantia nigra following 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) treatment of mice
3. Bax suppresses tumorigenesis and stimulates apoptosis in vivo
4. BAX-induced cell death may not require interleukin 1 -converting enzyme-like proteases
5. Cyclosporin and carnitine prevent the anoxic death of cultured hepatocytes by inhibiting the mitochondrial permeability transition
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