Mutagenesis Identifies New Signals for β-Amyloid Precursor Protein Endocytosis, Turnover, and the Generation of Secreted Fragments, Including Aβ42-Reference-Cited by-同舟云学术

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Mutagenesis Identifies New Signals for β-Amyloid Precursor Protein Endocytosis, Turnover, and the Generation of Secreted Fragments, Including Aβ42

Published:1999-07 Issue:27 Volume:274 Page:18851-18856
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Perez Ruth G.,Soriano Salvador,Hayes Jay D.,Ostaszewski Beth,Xia Weiming,Selkoe Dennis J.,Chen Xiaohua,Stokin Gorazd B.,Koo Edward H.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference56 articles.

1. The precursor of Alzheimer's disease amyloid A4 protein resembles a cell-surface receptor

2. Alzheimer's disease amyloidogenic glycoprotein: expression pattern in rat brain suggests a role in cell contact.

3. Beta amyloid precursor protein mediates neuronal cell-cell and cell-surface adhesion

4. Alzheimer's beta-amyloid precursor protein is expressed on the surface of immediately ex vivo brain cells: A flow cytometric study

5. Identification of the Alzheimer beta/A4 amyloid precursor protein in clathrin-coated vesicles purified from PC12 cells.

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